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  • Bromocriptine vs cabergoline pregnancy
    Posted Apr 28, 2017 by Admin

    Stimulating dopamine receptors reduces the production of the pituitary hormone prolactin, reduces the levels of growth hormone in people with acromegaly, and improves symptoms of Parkinson s. The FDA approved bromocriptine on June 28, 1978.Your doctor may start you on 0.375 mg and adjust your.

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Cabergoline blood sugar

Posted Mar 20, 2016 by Admin

METHODS This study was performed in 17 overweight women and men with type 2 diabetes with persistent hyperglycemia in spite of treatment with maximum dose of sulfonylurea, metformin and pioglitazone. 10 patients (group I) randomized to be treated with cabergoline 0.5 mg weekly for 3. Med J Malaysia. 2012; 67(4 390-2 (ISSN : ) Taghavi SM; Fatemi SS; Rokni H Mashhad Medical University, Endocrinology, Ahmad Abad, Mashhad, Khorasan Iran, Islamic Republic Of. UNLABELLED : Ergot-derived dopamine D2 receptor agonists are the usual treatment of hyperprolactinemia and Parkinson's disease and recently bromocriptine has been.

It reduces both fasting and postprandial plasma glucose levels and causes reduction in HbA1c. From MEDLINE /PubMed, a database of the U.S. National Library of Medicine.

Abstract Ergot-derived dopamine D2 receptor agonists are the usual treatment of hyperprolactinemia and Parkinson's disease and recently bromocriptine has been approved for the treatment of type 2 diabetes. The aim of this study was the evaluation of short-term effect of cabergoline in poorly controlled diabetic.

Bromocriptine patients cabergoline

RESULTS : FBS decreased from 210.70 /- 21.29 to 144.90 /- 26.56 mg/dl in cabergoline group whereas it decreased in placebo group insignificantly. Postprandial blood glucose decreased from 264.2 /- 28 mg/dl to 203.6 /- 34.34 mg/dl in cabergoline group whereas it increased in placebo.

Fasting and postprandial plasma glucose concentration and HbAlc measured in beginning and end of the study. RESULTS FBS decreased from 210.70 /- 21.29 to 144.90 /- 26.56 mg/dl in cabergoline group whereas it decreased in placebo group insignificantly.

10 patients (group I) randomized to be treated with cabergoline 0.5 mg weekly for 3 months and 7 patients (group II) with placebo. Fasting and postprandial plasma glucose concentration and HbAlc measured in beginning and end of the study.

Postprandial blood glucose decreased from 264.2 /- 28 mg/dl to 203.6 /- 34.34 mg/dl in cabergoline group whereas it increased in placebo group insignificantly. HbA1c decreased in cabergoline group from 8.48 /- 0.44 to 7.7 /- 0.11 whereas in control group it increased insignificantly from.